Heparin-Induced Renal Tubular Acidosis Masquerading as Hyperkalemia in a SARS-CoV-2 (COVID-19) Patient: A Case Report

Type 4 renal tubular acidosis (RTA) is a type of metabolic acidosis characterized by hyperchloremia and hyperkalemia resulting from the reduction in and/or resistance to aldosterone. RTA can be caused by multiple different medications including angiotensin-converting enzyme (ACE) inhibitor/angiotens...

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Bibliographic Details
Published in:Curēus (Palo Alto, CA) CA), 2021-12, Vol.13 (12), p.e20312
Main Authors: Baleguli, Vidya, Mahmood, Riaz, Herrera, Martin, Raybon-Rojas, Erine
Format: Article
Language:English
Subjects:
Acidosis
Anticoagulants
Blood
Case reports
Coronaviruses
COVID-19
Creatinine
Diuretics
Hydrogen
Hyperkalemia
Infectious Disease
Internal Medicine
Kidney diseases
Metabolism
Molecular weight
Nephrology
Nonsteroidal anti-inflammatory drugs
Patients
Pneumonia
Potassium
Severe acute respiratory syndrome coronavirus 2
Sodium
Thromboembolism
Tomography
Urine
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Summary:Type 4 renal tubular acidosis (RTA) is a type of metabolic acidosis characterized by hyperchloremia and hyperkalemia resulting from the reduction in and/or resistance to aldosterone. RTA can be caused by multiple different medications including angiotensin-converting enzyme (ACE) inhibitor/angiotensin receptor blocker (ARB), potassium-sparing diuretics, and heparin. In this case, we discuss renal tubular acidosis caused by heparin use for the prevention of thromboembolic disease in COVID-19 infections.
ISSN:2168-8184
2168-8184
DOI:10.7759/cureus.20312