Loading…

Aortic Knob Width: A Possible Marker of Vascular Remodeling in Obstructive Sleep Apnea

Obstructive sleep apnea (OSA) is characterized by recurrent episodes of upper airway collapse during sleep, resulting in apneas or hypopneas. Several cohort studies have recently shown that severe OSA is independently associated with an increased risk of myocardial infarction, stroke, and death from...

Full description

Saved in:
Bibliographic Details
Published in:Journal of Atherosclerosis and Thrombosis 2020/06/01, Vol.27(6), pp.499-500
Main Authors: Shiina, Kazuki, Tomiyama, Hirofumi, Takata, Yoshifumi, Chikamori, Taishiro
Format: Article
Language:eng ; jpn
Subjects:
Citations: Items that this one cites
Items that cite this one
Online Access:Get full text
Tags: Add Tag
No Tags, Be the first to tag this record!
cited_by cdi_FETCH-LOGICAL-c686t-18fb8d8c7bcfb2d64dcc31af4c540ba19b192b3c14dd01353e006c0f20b66043
cites cdi_FETCH-LOGICAL-c686t-18fb8d8c7bcfb2d64dcc31af4c540ba19b192b3c14dd01353e006c0f20b66043
container_end_page 500
container_issue 6
container_start_page 499
container_title Journal of Atherosclerosis and Thrombosis
container_volume 27
creator Shiina, Kazuki
Tomiyama, Hirofumi
Takata, Yoshifumi
Chikamori, Taishiro
description Obstructive sleep apnea (OSA) is characterized by recurrent episodes of upper airway collapse during sleep, resulting in apneas or hypopneas. Several cohort studies have recently shown that severe OSA is independently associated with an increased risk of myocardial infarction, stroke, and death from cardiovascular disease. The possible mechanisms underlying OSA contribution to atherosclerosis comprise complex and interrelated cascades. Therefore, it is difficult to elucidate the causal relationship between OSA and atherosclerosis. Among these mechanisms, intermittent hypoxia is believed to be the main component linking OSA to atherosclerosis.
doi_str_mv 10.5551/jat.ED123
format article
fullrecord <record><control><sourceid>proquest_pubme</sourceid><recordid>TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_7355104</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><sourcerecordid>2344226897</sourcerecordid><originalsourceid>FETCH-LOGICAL-c686t-18fb8d8c7bcfb2d64dcc31af4c540ba19b192b3c14dd01353e006c0f20b66043</originalsourceid><addsrcrecordid>eNpVUU1vEzEUXCEQLYUDfwD5CIcUf6-XAygqpSCKiqAqR8v2vk0cHDvYuxX99zhJieDy3pNmNG800zTPCT4VQpDXKzOenr8nlD1ojolSeMZUyx7Wm_F681YdNU9KWWHMmBD0cXPESCc7QdlxczNPefQOfY7Joh--H5dv0Bx9TaV4GwB9MfknZJQGdGOKm4LJ6BusUw_BxwXyEV3ZMubJjf4W0PcAsEHzTQTztHk0mFDg2f0-aa4_nF-ffZxdXl18OptfzpxUcpwRNVjVK9daN1jaS947x4gZuBMcW0M6SzpqmSO87zFhggHG0uGBYisl5uykebuX3Ux2Db2DOGYT9Cb7tcl3Ohmv_0eiX-pFutVtDYLsBF7eC-T0a4Iy6rUvDkIwEdJUNGWcUypV11bqqz3V5ZpOhuHwhmC9rUHXGvSuhsp98a-vA_Nv7pVwsSdU1DsTUqyBgl6lKceal4bfbZ_Wd0ZTTLHGmLZYbpfGvOs0FrVKoQijoiq92yutymgWcHhltrUG2JmirZbbsTN3QNzSZA2R_QFh-rE3</addsrcrecordid><sourcetype>Open Access Repository</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>2344226897</pqid></control><display><type>article</type><title>Aortic Knob Width: A Possible Marker of Vascular Remodeling in Obstructive Sleep Apnea</title><source>PubMed Central</source><source>EZB Electronic Journals Library</source><creator>Shiina, Kazuki ; Tomiyama, Hirofumi ; Takata, Yoshifumi ; Chikamori, Taishiro</creator><creatorcontrib>Shiina, Kazuki ; Tomiyama, Hirofumi ; Takata, Yoshifumi ; Chikamori, Taishiro ; Department of Cardiology ; Tokyo Medical University</creatorcontrib><description>Obstructive sleep apnea (OSA) is characterized by recurrent episodes of upper airway collapse during sleep, resulting in apneas or hypopneas. Several cohort studies have recently shown that severe OSA is independently associated with an increased risk of myocardial infarction, stroke, and death from cardiovascular disease. The possible mechanisms underlying OSA contribution to atherosclerosis comprise complex and interrelated cascades. Therefore, it is difficult to elucidate the causal relationship between OSA and atherosclerosis. Among these mechanisms, intermittent hypoxia is believed to be the main component linking OSA to atherosclerosis.</description><identifier>ISSN: 1340-3478</identifier><identifier>EISSN: 1880-3873</identifier><identifier>DOI: 10.5551/jat.ED123</identifier><identifier>PMID: 31969523</identifier><language>eng ; jpn</language><publisher>Japan: Japan Atherosclerosis Society</publisher><subject>Aorta - diagnostic imaging ; Aorta - pathology ; Correlation of Data ; Early Diagnosis ; Humans ; Organ Size ; Polysomnography - methods ; Radiography, Thoracic - methods ; Severity of Illness Index ; Sleep Apnea, Obstructive - diagnosis ; Sleep Apnea, Obstructive - physiopathology ; Vascular Remodeling ; Vascular Stiffness</subject><ispartof>Journal of Atherosclerosis and Thrombosis, 2020/06/01, Vol.27(6), pp.499-500</ispartof><rights>2020 This article is distributed under the terms of the latest version of CC BY-NC-SA defined by the Creative Commons Attribution License.</rights><rights>2020 Japan Atherosclerosis Society 2020</rights><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c686t-18fb8d8c7bcfb2d64dcc31af4c540ba19b192b3c14dd01353e006c0f20b66043</citedby><cites>FETCH-LOGICAL-c686t-18fb8d8c7bcfb2d64dcc31af4c540ba19b192b3c14dd01353e006c0f20b66043</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC7355104/pdf/$$EPDF$$P50$$Gpubmedcentral$$Hfree_for_read</linktopdf><linktohtml>$$Uhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC7355104/$$EHTML$$P50$$Gpubmedcentral$$Hfree_for_read</linktohtml><link.rule.ids>230,315,733,786,790,891,27957,27958,53827,53829</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/31969523$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Shiina, Kazuki</creatorcontrib><creatorcontrib>Tomiyama, Hirofumi</creatorcontrib><creatorcontrib>Takata, Yoshifumi</creatorcontrib><creatorcontrib>Chikamori, Taishiro</creatorcontrib><creatorcontrib>Department of Cardiology</creatorcontrib><creatorcontrib>Tokyo Medical University</creatorcontrib><title>Aortic Knob Width: A Possible Marker of Vascular Remodeling in Obstructive Sleep Apnea</title><title>Journal of Atherosclerosis and Thrombosis</title><addtitle>JAT</addtitle><description>Obstructive sleep apnea (OSA) is characterized by recurrent episodes of upper airway collapse during sleep, resulting in apneas or hypopneas. Several cohort studies have recently shown that severe OSA is independently associated with an increased risk of myocardial infarction, stroke, and death from cardiovascular disease. The possible mechanisms underlying OSA contribution to atherosclerosis comprise complex and interrelated cascades. Therefore, it is difficult to elucidate the causal relationship between OSA and atherosclerosis. Among these mechanisms, intermittent hypoxia is believed to be the main component linking OSA to atherosclerosis.</description><subject>Aorta - diagnostic imaging</subject><subject>Aorta - pathology</subject><subject>Correlation of Data</subject><subject>Early Diagnosis</subject><subject>Humans</subject><subject>Organ Size</subject><subject>Polysomnography - methods</subject><subject>Radiography, Thoracic - methods</subject><subject>Severity of Illness Index</subject><subject>Sleep Apnea, Obstructive - diagnosis</subject><subject>Sleep Apnea, Obstructive - physiopathology</subject><subject>Vascular Remodeling</subject><subject>Vascular Stiffness</subject><issn>1340-3478</issn><issn>1880-3873</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2020</creationdate><recordtype>article</recordtype><recordid>eNpVUU1vEzEUXCEQLYUDfwD5CIcUf6-XAygqpSCKiqAqR8v2vk0cHDvYuxX99zhJieDy3pNmNG800zTPCT4VQpDXKzOenr8nlD1ojolSeMZUyx7Wm_F681YdNU9KWWHMmBD0cXPESCc7QdlxczNPefQOfY7Joh--H5dv0Bx9TaV4GwB9MfknZJQGdGOKm4LJ6BusUw_BxwXyEV3ZMubJjf4W0PcAsEHzTQTztHk0mFDg2f0-aa4_nF-ffZxdXl18OptfzpxUcpwRNVjVK9daN1jaS947x4gZuBMcW0M6SzpqmSO87zFhggHG0uGBYisl5uykebuX3Ux2Db2DOGYT9Cb7tcl3Ohmv_0eiX-pFutVtDYLsBF7eC-T0a4Iy6rUvDkIwEdJUNGWcUypV11bqqz3V5ZpOhuHwhmC9rUHXGvSuhsp98a-vA_Nv7pVwsSdU1DsTUqyBgl6lKceal4bfbZ_Wd0ZTTLHGmLZYbpfGvOs0FrVKoQijoiq92yutymgWcHhltrUG2JmirZbbsTN3QNzSZA2R_QFh-rE3</recordid><startdate>20200601</startdate><enddate>20200601</enddate><creator>Shiina, Kazuki</creator><creator>Tomiyama, Hirofumi</creator><creator>Takata, Yoshifumi</creator><creator>Chikamori, Taishiro</creator><general>Japan Atherosclerosis Society</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope><scope>5PM</scope></search><sort><creationdate>20200601</creationdate><title>Aortic Knob Width: A Possible Marker of Vascular Remodeling in Obstructive Sleep Apnea</title><author>Shiina, Kazuki ; Tomiyama, Hirofumi ; Takata, Yoshifumi ; Chikamori, Taishiro</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c686t-18fb8d8c7bcfb2d64dcc31af4c540ba19b192b3c14dd01353e006c0f20b66043</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng ; jpn</language><creationdate>2020</creationdate><topic>Aorta - diagnostic imaging</topic><topic>Aorta - pathology</topic><topic>Correlation of Data</topic><topic>Early Diagnosis</topic><topic>Humans</topic><topic>Organ Size</topic><topic>Polysomnography - methods</topic><topic>Radiography, Thoracic - methods</topic><topic>Severity of Illness Index</topic><topic>Sleep Apnea, Obstructive - diagnosis</topic><topic>Sleep Apnea, Obstructive - physiopathology</topic><topic>Vascular Remodeling</topic><topic>Vascular Stiffness</topic><toplevel>online_resources</toplevel><creatorcontrib>Shiina, Kazuki</creatorcontrib><creatorcontrib>Tomiyama, Hirofumi</creatorcontrib><creatorcontrib>Takata, Yoshifumi</creatorcontrib><creatorcontrib>Chikamori, Taishiro</creatorcontrib><creatorcontrib>Department of Cardiology</creatorcontrib><creatorcontrib>Tokyo Medical University</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><collection>PubMed Central (Full Participant titles)</collection><jtitle>Journal of Atherosclerosis and Thrombosis</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Shiina, Kazuki</au><au>Tomiyama, Hirofumi</au><au>Takata, Yoshifumi</au><au>Chikamori, Taishiro</au><aucorp>Department of Cardiology</aucorp><aucorp>Tokyo Medical University</aucorp><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Aortic Knob Width: A Possible Marker of Vascular Remodeling in Obstructive Sleep Apnea</atitle><jtitle>Journal of Atherosclerosis and Thrombosis</jtitle><addtitle>JAT</addtitle><date>2020-06-01</date><risdate>2020</risdate><volume>27</volume><issue>6</issue><spage>499</spage><epage>500</epage><pages>499-500</pages><issn>1340-3478</issn><eissn>1880-3873</eissn><notes>SourceType-Other Sources-1</notes><notes>content type line 63</notes><notes>ObjectType-Editorial-2</notes><notes>ObjectType-Commentary-1</notes><abstract>Obstructive sleep apnea (OSA) is characterized by recurrent episodes of upper airway collapse during sleep, resulting in apneas or hypopneas. Several cohort studies have recently shown that severe OSA is independently associated with an increased risk of myocardial infarction, stroke, and death from cardiovascular disease. The possible mechanisms underlying OSA contribution to atherosclerosis comprise complex and interrelated cascades. Therefore, it is difficult to elucidate the causal relationship between OSA and atherosclerosis. Among these mechanisms, intermittent hypoxia is believed to be the main component linking OSA to atherosclerosis.</abstract><cop>Japan</cop><pub>Japan Atherosclerosis Society</pub><pmid>31969523</pmid><doi>10.5551/jat.ED123</doi><tpages>2</tpages><oa>free_for_read</oa></addata></record>
fulltext fulltext
identifier ISSN: 1340-3478
ispartof Journal of Atherosclerosis and Thrombosis, 2020/06/01, Vol.27(6), pp.499-500
issn 1340-3478
1880-3873
language eng ; jpn
recordid cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_7355104
source PubMed Central; EZB Electronic Journals Library
subjects Aorta - diagnostic imaging
Aorta - pathology
Correlation of Data
Early Diagnosis
Humans
Organ Size
Polysomnography - methods
Radiography, Thoracic - methods
Severity of Illness Index
Sleep Apnea, Obstructive - diagnosis
Sleep Apnea, Obstructive - physiopathology
Vascular Remodeling
Vascular Stiffness
title Aortic Knob Width: A Possible Marker of Vascular Remodeling in Obstructive Sleep Apnea
url http://sfxeu10.hosted.exlibrisgroup.com/loughborough?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2024-09-22T06%3A38%3A59IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-proquest_pubme&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=Aortic%20Knob%20Width:%20A%20Possible%20Marker%20of%20Vascular%20Remodeling%20in%20Obstructive%20Sleep%20Apnea&rft.jtitle=Journal%20of%20Atherosclerosis%20and%20Thrombosis&rft.au=Shiina,%20Kazuki&rft.aucorp=Department%20of%20Cardiology&rft.date=2020-06-01&rft.volume=27&rft.issue=6&rft.spage=499&rft.epage=500&rft.pages=499-500&rft.issn=1340-3478&rft.eissn=1880-3873&rft_id=info:doi/10.5551/jat.ED123&rft_dat=%3Cproquest_pubme%3E2344226897%3C/proquest_pubme%3E%3Cgrp_id%3Ecdi_FETCH-LOGICAL-c686t-18fb8d8c7bcfb2d64dcc31af4c540ba19b192b3c14dd01353e006c0f20b66043%3C/grp_id%3E%3Coa%3E%3C/oa%3E%3Curl%3E%3C/url%3E&rft_id=info:oai/&rft_pqid=2344226897&rft_id=info:pmid/31969523&rfr_iscdi=true