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Aortic Knob Width: A Possible Marker of Vascular Remodeling in Obstructive Sleep Apnea
Obstructive sleep apnea (OSA) is characterized by recurrent episodes of upper airway collapse during sleep, resulting in apneas or hypopneas. Several cohort studies have recently shown that severe OSA is independently associated with an increased risk of myocardial infarction, stroke, and death from...
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Published in: | Journal of Atherosclerosis and Thrombosis 2020/06/01, Vol.27(6), pp.499-500 |
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container_title | Journal of Atherosclerosis and Thrombosis |
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creator | Shiina, Kazuki Tomiyama, Hirofumi Takata, Yoshifumi Chikamori, Taishiro |
description | Obstructive sleep apnea (OSA) is characterized by recurrent episodes of upper airway collapse during sleep, resulting in apneas or hypopneas. Several cohort studies have recently shown that severe OSA is independently associated with an increased risk of myocardial infarction, stroke, and death from cardiovascular disease. The possible mechanisms underlying OSA contribution to atherosclerosis comprise complex and interrelated cascades. Therefore, it is difficult to elucidate the causal relationship between OSA and atherosclerosis. Among these mechanisms, intermittent hypoxia is believed to be the main component linking OSA to atherosclerosis. |
doi_str_mv | 10.5551/jat.ED123 |
format | article |
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Among these mechanisms, intermittent hypoxia is believed to be the main component linking OSA to atherosclerosis.</description><subject>Aorta - diagnostic imaging</subject><subject>Aorta - pathology</subject><subject>Correlation of Data</subject><subject>Early Diagnosis</subject><subject>Humans</subject><subject>Organ Size</subject><subject>Polysomnography - methods</subject><subject>Radiography, Thoracic - methods</subject><subject>Severity of Illness Index</subject><subject>Sleep Apnea, Obstructive - diagnosis</subject><subject>Sleep Apnea, Obstructive - physiopathology</subject><subject>Vascular Remodeling</subject><subject>Vascular Stiffness</subject><issn>1340-3478</issn><issn>1880-3873</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2020</creationdate><recordtype>article</recordtype><recordid>eNpVUU1vEzEUXCEQLYUDfwD5CIcUf6-XAygqpSCKiqAqR8v2vk0cHDvYuxX99zhJieDy3pNmNG800zTPCT4VQpDXKzOenr8nlD1ojolSeMZUyx7Wm_F681YdNU9KWWHMmBD0cXPESCc7QdlxczNPefQOfY7Joh--H5dv0Bx9TaV4GwB9MfknZJQGdGOKm4LJ6BusUw_BxwXyEV3ZMubJjf4W0PcAsEHzTQTztHk0mFDg2f0-aa4_nF-ffZxdXl18OptfzpxUcpwRNVjVK9daN1jaS947x4gZuBMcW0M6SzpqmSO87zFhggHG0uGBYisl5uykebuX3Ux2Db2DOGYT9Cb7tcl3Ohmv_0eiX-pFutVtDYLsBF7eC-T0a4Iy6rUvDkIwEdJUNGWcUypV11bqqz3V5ZpOhuHwhmC9rUHXGvSuhsp98a-vA_Nv7pVwsSdU1DsTUqyBgl6lKceal4bfbZ_Wd0ZTTLHGmLZYbpfGvOs0FrVKoQijoiq92yutymgWcHhltrUG2JmirZbbsTN3QNzSZA2R_QFh-rE3</recordid><startdate>20200601</startdate><enddate>20200601</enddate><creator>Shiina, Kazuki</creator><creator>Tomiyama, Hirofumi</creator><creator>Takata, Yoshifumi</creator><creator>Chikamori, Taishiro</creator><general>Japan Atherosclerosis Society</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope><scope>5PM</scope></search><sort><creationdate>20200601</creationdate><title>Aortic Knob Width: A Possible Marker of Vascular Remodeling in Obstructive Sleep Apnea</title><author>Shiina, Kazuki ; Tomiyama, Hirofumi ; Takata, Yoshifumi ; Chikamori, Taishiro</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c686t-18fb8d8c7bcfb2d64dcc31af4c540ba19b192b3c14dd01353e006c0f20b66043</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng ; jpn</language><creationdate>2020</creationdate><topic>Aorta - diagnostic imaging</topic><topic>Aorta - pathology</topic><topic>Correlation of Data</topic><topic>Early Diagnosis</topic><topic>Humans</topic><topic>Organ Size</topic><topic>Polysomnography - methods</topic><topic>Radiography, Thoracic - methods</topic><topic>Severity of Illness Index</topic><topic>Sleep Apnea, Obstructive - diagnosis</topic><topic>Sleep Apnea, Obstructive - physiopathology</topic><topic>Vascular Remodeling</topic><topic>Vascular Stiffness</topic><toplevel>online_resources</toplevel><creatorcontrib>Shiina, Kazuki</creatorcontrib><creatorcontrib>Tomiyama, Hirofumi</creatorcontrib><creatorcontrib>Takata, Yoshifumi</creatorcontrib><creatorcontrib>Chikamori, Taishiro</creatorcontrib><creatorcontrib>Department of Cardiology</creatorcontrib><creatorcontrib>Tokyo Medical University</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><collection>PubMed Central (Full Participant titles)</collection><jtitle>Journal of Atherosclerosis and Thrombosis</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Shiina, Kazuki</au><au>Tomiyama, Hirofumi</au><au>Takata, Yoshifumi</au><au>Chikamori, Taishiro</au><aucorp>Department of Cardiology</aucorp><aucorp>Tokyo Medical University</aucorp><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Aortic Knob Width: A Possible Marker of Vascular Remodeling in Obstructive Sleep Apnea</atitle><jtitle>Journal of Atherosclerosis and Thrombosis</jtitle><addtitle>JAT</addtitle><date>2020-06-01</date><risdate>2020</risdate><volume>27</volume><issue>6</issue><spage>499</spage><epage>500</epage><pages>499-500</pages><issn>1340-3478</issn><eissn>1880-3873</eissn><notes>SourceType-Other Sources-1</notes><notes>content type line 63</notes><notes>ObjectType-Editorial-2</notes><notes>ObjectType-Commentary-1</notes><abstract>Obstructive sleep apnea (OSA) is characterized by recurrent episodes of upper airway collapse during sleep, resulting in apneas or hypopneas. Several cohort studies have recently shown that severe OSA is independently associated with an increased risk of myocardial infarction, stroke, and death from cardiovascular disease. The possible mechanisms underlying OSA contribution to atherosclerosis comprise complex and interrelated cascades. Therefore, it is difficult to elucidate the causal relationship between OSA and atherosclerosis. Among these mechanisms, intermittent hypoxia is believed to be the main component linking OSA to atherosclerosis.</abstract><cop>Japan</cop><pub>Japan Atherosclerosis Society</pub><pmid>31969523</pmid><doi>10.5551/jat.ED123</doi><tpages>2</tpages><oa>free_for_read</oa></addata></record> |
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source | PubMed Central; EZB Electronic Journals Library |
subjects | Aorta - diagnostic imaging Aorta - pathology Correlation of Data Early Diagnosis Humans Organ Size Polysomnography - methods Radiography, Thoracic - methods Severity of Illness Index Sleep Apnea, Obstructive - diagnosis Sleep Apnea, Obstructive - physiopathology Vascular Remodeling Vascular Stiffness |
title | Aortic Knob Width: A Possible Marker of Vascular Remodeling in Obstructive Sleep Apnea |
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