AKT and the Phosphatidylinositol 3-Kinase/AKT Pathway: Important Molecular Targets for Lung Cancer Prevention and Treatment

[Chun KH] et al. (1) derived several cell lines from simian virus 40-immortalized human bronchial epithelial (HBE) cells. These cell lines represented premalignant and malignant HBE cells and were evaluated along with normal HBE (NHBE) cells.

Saved in:
Bibliographic Details
Published in:JNCI : Journal of the National Cancer Institute 2003-02, Vol.95 (4), p.252-253
Main Authors: Crowell, James A., Steele, Vernon E.
Format: Article
Language:English
Subjects:
Antineoplastic Agents - pharmacology
Cell Cycle - drug effects
Cells
Disease control
Disease management
Humans
Lung cancer
Lung Neoplasms - drug therapy
Lung Neoplasms - metabolism
Lung Neoplasms - prevention & control
Phosphatidylinositol 3-Kinases - drug effects
Phosphatidylinositol 3-Kinases - metabolism
Phosphorylation - drug effects
Protein-Serine-Threonine Kinases
Proto-Oncogene Proteins - drug effects
Proto-Oncogene Proteins - metabolism
Proto-Oncogene Proteins c-akt
Rotenone - analogs & derivatives
Rotenone - pharmacology
Signal Transduction - drug effects
Tumor Cells, Cultured
Citations: Items that this one cites
Items that cite this one
Online Access:Get full text
Tags: Add Tag
No Tags, Be the first to tag this record!
Description
Summary:[Chun KH] et al. (1) derived several cell lines from simian virus 40-immortalized human bronchial epithelial (HBE) cells. These cell lines represented premalignant and malignant HBE cells and were evaluated along with normal HBE (NHBE) cells.
ISSN:0027-8874
1460-2105
DOI:10.1093/jnci/95.4.252